TBI Treatment and Management in Nursing Care

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Introduction

Assessments are the foundation to ensure no injury or abnormality is overlooked. Trauma centers across the county utilize a systematic method to ensure this is done.  The following case study will review the elements used in the Trauma Nurse Core Course (TNCC) to ensure no injuries are overlooked for this patient.   

The Centers for Disease Control and Prevention (CDC) report more than 32,000 people are killed in MVCs and 2 million are injured each year from an MVC (4). Identification and thorough assessments can lead to successful TBI treatment plans, and it is of the utmost importance for trauma nurses to remain up to date on the current best practices. TBIs are commonly seen in patients after an MVC, the following case study will walk you through the assessment, physician orders, diagnostics, TBI treatment, and outcome for an MVC patient.  

Case Study and Trauma Nurse Assessment for Potential TBI Treatment 

A 46-year-old man presents to the emergency department (ED) after an MVC.  He was a restrained passenger, +airbag deployment, and no reported loss of consciousness. The accident occurred when another car failed to stop at a traffic light and hit the victim‘s car on the passenger side. EMS reports heavy damage and difficult extraction. The victim is complaining of a headache, nausea, and R shoulder pain. Below, is your complete trauma nurse assessment: 

CDC TBI Fast Facts  

All of the following are from a 2014 CDC report: 

• About 2.87 million TBI-related emergency department (ED) visits, hospitalizations, and deaths occurred in the United States, including over 837,000 of these health events among children. 
• TBI contributed to the deaths of 56,800 people, including 2,529 deaths among children. 
• TBI was diagnosed in approximately 288,000 hospitalizations, including over 23,000 among children, consisting of either TBI alone or TBI with another injury. 
• An average of 155 people in the United States died each day from injuries that include a TBI (4).

Case Study (cont.) 

Time elapsed since arrival: 3 hours

Labs: CBC, CMP 

– Chest x-ray, C-Spine, R Shoulder  

– CT-head  

– Wound care per protocol 

– Tdap 0.5ml IM NOW 

– EKG 

– Zofran 4mg IVP NOW 

Nursing Assessment:  

• Patient is alert to name 
• Stated he was at another local ED,  
• Stated wrong year  
• Vital signs:  

– Temp 97.7

– Heart rate 108 

– Resp 16 

– BP 148/82 

– SPO2 95%  

• Complaining of pain R shoulder (6/10) 
• Headache (9/10)  
• C/o of nausea and feeling dizzy. 
• GCS=14 
• Also noted L hand grasp had declined from 5/5 to 3/5, and R/L lower extremities 4/5.  

The following labs/radiology results are back: 

• CBC- results WNL expect for WBC 14,000/mL and platelets 135,000/mL 
• CMP- WNL 
• PT/PTT- WNL 
• CT Head Report- Abnormal hyperdense area of blood noted along the R cerebral convexity, crescent–shaped from the fronto-temporo to parietal area. These are positive findings for a subdural hematoma.  
• C-Spine– All cervical spine bodies and intervertebral spaces in correct alignment, no noted cervical spine abnormalities. 
• R shoulder– inferior subluxation of R humeral head  

Subdural Hematoma

A subdural hematoma (SDH) forms when there is a venous hemorrhage into the space between the dura and the arachnoid membranes. The brain has three layers or coverings (called meninges) that lay between the bony skull and the actual brain tissue. The layers from the skull to the brain are the dura mater, arachnoid mater, and pia mater. The purpose of the meninges is to cover and protect the brain.  

The blood from an SDH is usually the result of damage to the cerebral veins as they empty into the dural venous sinuses (1).  The bleeding from an SDH is under the skull and outside the brain, not in the brain itself. As the bleeding continues, it puts increased pressure on the brain, which usually causes symptoms.  As the pressure in the skull rises to a high level, a subdural hematoma can lead to unconsciousness and death (1,2,3). 

Causes

A subdural hematoma is usually caused by a head injury, such as a fall, MVC, or an assault. The sudden blow to the head tears blood vessels that run along the surface of the brain. This is referred to as an acute SDH and occur in up to 25% of people who have experienced a head injury.  

Types

• Acute: Most dangerous. The symptoms are usually severe and appear right after a head injury, often within minutes to hours. As blood develops, the pressure on the brain increases, and if not identified and treated quickly, a person can lose consciousness, become paralyzed or death can occur. 
• Subacute: The symptoms can appear from many hours to even weeks after the head injury 
• Chronic: This is most common in older people, the bleeding is slow and symptoms are often vague.  It could be caused by even a minor head injury that often the person does not remember (1,2).  

Symptoms

Symptoms are mostly based on the rate of bleeding (1): 

• Headache– that doesn‘t go away 
• Confusion or drowsiness 
• Change in behavior 
• Dizziness, loss of balance  
• Nausea and vomiting 
• Lethargy or excessive drowsiness 
• Weakness 
• Apathy 

If not identified and bleeding progresses, symptoms like seizures, paralysis, breathing issues, +LOC, or coma can occur. 

Other things that influence SDH are age, other medical conditions, medications that influence clotting.   

Diagnosing and Treating SDH 

A computed tomography (CT) scan or magnetic resonance imaging (MRI) are the two diagnostic interventions that are usually ordered to outline any potential TBI treatment (3).  

Treatment of SDH depends on the patient severity and can range from watchful waiting to brain surgery. 

If a patient has a small SDH with mild symptoms, the provider may recommend close observation and a repeated CT of the head to assess for changes.  

More severe SDHs require surgery to reduce the pressure on the brain. In general, the surgeons will use one of these three techniques to treat SDHs (1): 

• Burr hole trephination 
• Craniotomy 
• Craniectomy 

Case Study (cont.) 

Time elapsed since arrival: 4.5 hours  

Nursing Assessment:  

• Patient is alert to name and wife 
• Unable to state year or place 
• Vital signs: 

– Temp: 97.7 

– Heart rate: 118 

– Resp: 14 

– BP: 158/88 

– SPO2 95%  

• Complaining of R shoulder pain (5/10)  
• Headache (10/10) 
• Nausea continues.
  
• GCS=14 
• No change in motor function from prior.
  
• Pt is acting very sleepy and is having a hard time focusing on questions. 
  

Physician Update:

• Neurologist consult obtained and pt is being prepped for OR.
  
• Surgical Checklist completed.

Time elapsed since arrival: 24 hours  

The patient is now in the Surgical ICU and is expected to go home tomorrow; the surgeon was able to evacuate the SDH using the Burr hole method. 

• His mental status is now at baseline, and the patient only has a mild headache (2/10).  
• Vital signs are all WNL.   
• The laceration was cleansed, and derma bound was used to close.  
• The R shoulder subluxation was positioned in correct alignment, and a repeat x-ray was taken to ensure alignment, then a shoulder immobilizer was placed on the patient; it should heal with 4-6 weeks of limited movement.  

Differentiating SDH from Epidural Hematomas 

An epidural hematoma is an abnormal collection of blood above the dura mater, so it is located between the skull and dura mater. The middle meningeal artery is the main artery nourishing the skull and usually the causative agent in an epidural bleed; as with all arteries, it is a high-pressure system, not low like the subdural bleed and venous system.  

Epidural hematomas are mostly caused by blunt trauma, especially to the temporal region, a thinner area. Patients with falls or skull fractures must always be assessed for TBI, and one should question any change in mental status as significant.  Epidural bleeds to carry some common signs and symptoms which include: 

• Age, usually < 45
• A fall or blunt trauma has occurred with a brief loss of consciousness, followed by a period of the patient feeling normal then getting drowsy 2-3 later (1,2). 
• Nausea with vomiting and a headache (1,2).  
• Head CT shows a convex or lens shape area where the bleed is located.  
• TBI treatment is like the SDH; the size and symptoms are key drivers; the pressure must be relieved from the brain to ensure the patient does not have permanent damage.  

Conclusion 

A systematic and detailed nursing assessment is critical in identifying any and all changes with patients; as you have learned many times, there are similarities with TBI. In order to provide the best TBI treatment plans to your patients, it is important to ask the right questions and complete systematic neurological assessments. Proper TBI treatment can truly make all the difference in your patient’s recovery.  

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